Proteins and Enzymes

A-Level Biology · Biological Molecules

Proteins

Proteins are polymers of amino acids. There are 20 common amino acids, each with the same basic structure — a central carbon bonded to an amino group (–NH₂), a carboxyl group (–COOH), a hydrogen, and a variable R group that gives each amino acid its properties.

Amino acids join by condensation, forming a peptide bond (releasing water) to make dipeptides and polypeptides.

The four levels of protein structure

LevelDescription
PrimaryThe sequence of amino acids in the polypeptide chain (determined by the gene)
SecondaryLocal folding into α-helices and β-pleated sheets, held by hydrogen bonds
TertiaryThe overall 3D shape of the chain, held by hydrogen bonds, ionic bonds, disulfide bridges and hydrophobic interactions
QuaternaryTwo or more polypeptide chains joined together (e.g. haemoglobin has 4 chains)

The precise 3D shape determines a protein's function — especially for enzymes and receptors.

Types of protein

  • Fibrous (e.g. collagen) — long, structural, insoluble.
  • Globular (e.g. enzymes, haemoglobin) — rounded, soluble, functional.

Test for proteins: the Biuret test — add Biuret reagent → turns purple/lilac if protein is present.

Enzymes

Enzymes are globular proteins that act as biological catalysts, lowering the activation energy of reactions. The substrate binds to the enzyme's active site.

  • Lock and key model: the substrate fits a rigid, complementary active site.
  • Induced fit model (more accurate): the active site changes shape slightly to mould around the substrate, straining bonds and lowering activation energy further.

Factors affecting enzyme activity

  • Temperature: rate rises to an optimum, then the enzyme denatures (tertiary structure/active site destroyed).
  • pH: each enzyme has an optimum pH; extremes denature it.
  • Substrate concentration: rate rises until all active sites are saturated (Vmax).
  • Inhibitors: competitive inhibitors bind the active site (overcome by more substrate); non-competitive inhibitors bind elsewhere, changing the active site's shape (not overcome by more substrate).

Worked example

Why does a change in a single amino acid (primary structure) alter a protein's function?

  • The primary sequence determines how the chain folds, so one change can alter the hydrogen/ionic/disulfide bonds in the tertiary structure, changing the 3D shape — and for an enzyme, its active site. ✓

Common mistakes

  • Confusing the bonds at each level (secondary = H-bonds; tertiary adds ionic, disulfide, hydrophobic).
  • Saying enzymes are "used up" — they are catalysts and are reused.
  • Mixing up competitive (active site) vs non-competitive (elsewhere) inhibition.

Exam tips

  • Learn the four structure levels and the bonds holding each.
  • Explain enzyme action via induced fit and lowering activation energy.
  • Distinguish competitive vs non-competitive inhibitors, including the effect of adding more substrate.

Key facts to remember

  • Proteins = amino acids joined by peptide bonds; structure levels primary → secondary (H-bonds) → tertiary (ionic/disulfide/hydrophobic) → quaternary.
  • 3D shape determines function; the Biuret test goes purple for protein.
  • Enzymes lower activation energy via induced fit; affected by temperature, pH, substrate concentration and competitive/non-competitive inhibitors.
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